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Emergency · Acute ScrotumEmergency / Scrotum

Testicular Torsion

The twisting of the testis on the spermatic cord strangles its own blood supply — a surgical emergency where salvage depends on time, so a clinically obvious torsion goes straight to exploration without waiting for imaging.

Assume
torsion
+
Window
~6 hours
+
Fix
both testes
Orientation

The big picture

Testicular torsion is the twisting of the testis on the spermatic cord, which strangles its own blood supply — a true surgical emergency where the testis dies by the hour. The entire approach is built around one fact: salvage depends on time. A torsed testis must be detorsed and fixed urgently; imaging must never delay surgery when the diagnosis is clinically obvious. It is the can't-miss diagnosis of the acute scrotum, especially in adolescents.

Golden rule

If torsion is clinically obvious, go straight to surgical exploration — do not delay for imaging.

Safety

Red flags

Sudden, severe unilateral scrotal/testicular pain

Can wake from sleep, occur at rest or after activity, often with nausea/vomiting.

High-riding, transversely-lying testis with a negative (absent) cremasteric reflex

On the affected side.

Acute onset — versus the more gradual onset of epididymo-orchitis

Mainly adolescents/young men, but can occur at any age (including neonates).

Pathophysiology

Mechanism pathway

Tap any step to see why it happens.

Illustration

Testicular torsion — Normal / Bell-clapper / Torsion

Testicular torsion — anatomy & the strangled blood supply

Normally the testis is anchored within the tunica vaginalis. In the bell-clapper deformity the tunica invests the cord high up, so the testis hangs free and can rotate. When it twists, the spermatic cord (and its vessels) are strangled — the testis loses its blood supply and infarcts.

Normal testis anchored, cord straight fixed to wall Bell-clapper deformity tunica invests high — testis hangs free horizontal lie · no fixation Torsion cord twists → blood supply strangled high-riding · ischaemic Twistedspermatic cord Ischaemictestis (infarcting)
Time is testis. Salvage is high if detorsed within ~6 hours and falls steeply after — each ~10 minutes of delay raises the chance of orchiectomy by ~4.8%. If torsion is clinically obvious, go straight to surgical exploration — don't delay for imaging.
Torsion twists the testis on the spermatic cord, strangling its own blood supply. The predisposing anatomy is the bell-clapper deformity (tunica vaginalis invests the cord high, so the testis hangs freely and can rotate) — present in ~12% of males and usually bilateral, which is why both testes are fixed at surgery. Management is emergency exploration: detorse, assess viability, then orchidopexy if viable / orchiectomy if not, plus contralateral fixation. Color Doppler (reduced/absent flow) helps only when the picture is equivocal and must not delay surgery. Grounded in leading urology references.
Work-up

Diagnostic algorithm

Each step answers one question. Tap to expand.

Management

Treatment ladder

1
Emergency surgical exploration — the definitive treatment

Detorse the testis, assess viability, and if viable, perform orchidopexy (fixation) — and fix the contralateral testis in the same sitting (the bell-clapper deformity is usually bilateral). A non-viable testis is removed (orchiectomy).

2
Manual detorsion — a temporising bridge only

Can be attempted as a temporising measure while preparing for theatre (classically "opening a book" — rotating the testis outward/laterally) — but it does not replace surgical fixation.

3
The clock is everything

Salvage is high if detorsed within ~6 hours, and falls steeply after. Each 10 minutes of delay increases the chance of orchiectomy by ~4.8%, and orchiectomy occurs in 30–70% at exploration in large series. Salvaged testes had on average less rotation than those requiring orchiectomy.

Exam

Board traps

Time is testis: salvage is high within ~6 hours and falls fast — each ~10 min of delay raises orchiectomy risk ~4.8%.

Don't delay surgery for imaging when torsion is clinically obvious.

Color Doppler shows REDUCED/ABSENT flow in torsion — the opposite of epididymo-orchitis (increased flow).

Bell-clapper deformity is usually bilateral → always fix BOTH testes at exploration.

Absent cremasteric reflex + high-riding, transverse testis point to torsion.

Neonatal torsion is extravaginal (the whole cord/tunica twists); the adolescent type is intravaginal.

Test yourself

Quiz

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